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In nerves uncovered to 100 Hz when bathed in 10 mM glucose, the CAP failed suggesting that even in the presence of normoglycemic glucose additional power substrate is required when the tissue vitality demand will increase. Such data are vital as they show that glycogen has an important role beneath physiological circumstances. The fact that glycogen supported CAP conduction in the absence of exogenously applied glucose probably has no value in ascribing a role to glycogen, since aglycaemia is an synthetic experimental process, and hypoglycemia is an iatrogenic consequence of insulin therapy that did not exist previous to 1922 (Frier et al., 2014), thus can't be glycogen’s important function. However supplying lactate to axons when tissue power demand will increase implies a novel function for mind glycogen and suggests it acts as an vitality buffer slightly than an power reserve, whose position is to not single-handedly help conduction, however to act in live performance with ambient ranges of normoglycemic glucose.

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